Do you want to know how muscles work?
Introducing video:
How our muscles work
Muscle contraction is produced by an interaction of the protein molecules of actin and myosin, which in turn occurs due to the sliding between thick and thin myofilaments, this sliding causes a decrease in the amplitude of band A and zone H.
Calcium (Ca) is the link between excitement and contraction of a muscle. The sarcoplasmic reticulum is specialized for its storage and release of calcium through the T system. When a nerve signal arrives at the muscle cell, a release of calcium is initiated into the fluid surrounding the filaments from special storage vesicles in the reticulum Sarcoplasmic, when combined with troponin, initiates the elimination of an impediment for a potential interaction between the actin and myosin filaments.
In any case, the tropomyosin, which in the resting muscle has turned off the active sites of actin, can now activate them; In turn the actin can activate the ATP hydrolysis of the myosin heads. This allows the myosin heads to attach to the actin filament sites. A bonded bridge during its action exerts a longitudinal force at a certain distance at which ATP is hydrolyzed and this attracts the actin filament along the center of the band A. This system shuts off when calcium is reuptake By the sarcoplasmic reticulum (the muscle relaxation effect of ATP is observed here, because the active transport of calcium to the sarcoplasmic reticulum is done at the expense of ATP), in the absence of calcium, the troponin-tropomyosin complex again prevents interaction between the Filaments actin and myosin, here the muscle fiber goes to rest state or cycle repeats again.
These events are repeated as the muscle is stimulated and the bridges are joined, rotated and cyclically detached, pushing the thin filaments beyond the thickness, shortening the muscle. The active site of the actin can react successively with various groups of linearly arranged myosin and the actin filament vanishes along the myosin filament.
How our muscles work
Muscle contraction is produced by an interaction of the protein molecules of actin and myosin, which in turn occurs due to the sliding between thick and thin myofilaments, this sliding causes a decrease in the amplitude of band A and zone H.
Calcium (Ca) is the link between excitement and contraction of a muscle. The sarcoplasmic reticulum is specialized for its storage and release of calcium through the T system. When a nerve signal arrives at the muscle cell, a release of calcium is initiated into the fluid surrounding the filaments from special storage vesicles in the reticulum Sarcoplasmic, when combined with troponin, initiates the elimination of an impediment for a potential interaction between the actin and myosin filaments.
In any case, the tropomyosin, which in the resting muscle has turned off the active sites of actin, can now activate them; In turn the actin can activate the ATP hydrolysis of the myosin heads. This allows the myosin heads to attach to the actin filament sites. A bonded bridge during its action exerts a longitudinal force at a certain distance at which ATP is hydrolyzed and this attracts the actin filament along the center of the band A. This system shuts off when calcium is reuptake By the sarcoplasmic reticulum (the muscle relaxation effect of ATP is observed here, because the active transport of calcium to the sarcoplasmic reticulum is done at the expense of ATP), in the absence of calcium, the troponin-tropomyosin complex again prevents interaction between the Filaments actin and myosin, here the muscle fiber goes to rest state or cycle repeats again.These events are repeated as the muscle is stimulated and the bridges are joined, rotated and cyclically detached, pushing the thin filaments beyond the thickness, shortening the muscle. The active site of the actin can react successively with various groups of linearly arranged myosin and the actin filament vanishes along the myosin filament.
Grear job!!!
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